Cardiac amyloidosis, a type of heart failure, was long considered a death sentence, but new medications are giving patients hope. When amyloid proteins build up in the heart, they make the muscle stiff and pumping blood more difficult. New research shows that drugs targeting transthyretin, one of the proteins that can cause cardiac amyloidosis, stop production before it can penetrate the heart. This "freezes" patients at their stage of heart failure, making early detection still key, said The New York Times.